Covid-19 – an acute respiratory infection caused by a new strain of corona virus characterized by symptoms similar to the flu. A healthy person becomes infected with the virus after breathing droplets from a cough or sneeze.
Subsequently, the silage incubation process begins when the virus is negative in the airways. Symptoms that followed include cough, fever, shortness of breath, headache, runny nose. But how exactly can Covid-19 progress, to put a man who is just running a nose into a ventilator and sometimes kill them?
For most patients, the corona virus begins to infect and cause the most severe damage inside their lungs. After all, like flu, Covid-19 is a respiratory disease.
The virus is inhaled from droplets shot by an infected person, when they cough or sneeze. From there, it will invade the airways of a healthy person and reach the lungs. In the lungs, Covid-19 found their target cells.
Like the previous SARS virus, Covid-19 has thorny proteins to bind to the ACE2 receptor. That receptor is only found in human lung and heart cells. But both strains of the virus release their hearts, for some reason scientists still don’t know.
Matthew B. Frieman, an associate professor at the University of Maryland who specializes in the study of highly toxic corona virus strains, said: Basically, Covid-19 will do what similar SARS virus did. It will cause disease in three stages: multiplying, stimulating excessive immune response and eventually hunting the lungs of the patient.
However, not all patients infected with the coronary virus end up with a decayed lung. Previous statistics show that only 25% of patients infected with SARS have severe respiratory failure. Similarly, new data shows that about 82% of Covid-19 infections exhibit only mild symptoms.
But in the case of a serious case? The corona virus will quickly colonize human lung cells through “card swipe port“ACE2. Lung cells come in two types: mucus-producing cells and hair-like cells called microcapsules.
Mucus is an essential component of the lungs, it protects lung tissue from pathogens and ensures that your respiratory organs do not dry out. Microscopic cells are dipped in mucus, they move constantly to clean up the strange debris that is inhaled in the air, such as pollen or virus.
Frieman explained that SARS loves to infect and destroy these microcapsules. The virus inserts its RNA into the lung cell DNA and takes control of it. Here, it will take advantage of the micro-cell’s protein and genetic material manufacturing machinery to synthesize and multiply thousands of times.
When the lung cell contains too many viruses, it simply explodes, releasing tens of thousands of viruses so that they continue to infect new cells. The chain reaction causes the microcapsules in the lungs of the patient to peel.
Patients will cough up them, along with mucus, debris, dirt and millions of Covid-19 viruses, which have just been replicated but have yet to enter new lung cells. This causes a range of symptoms including fever, cough, shortness of breath and pneumonia.
Stage one ends when the inflammatory process begins to form. At this point, the second stage begins when the immune system detects damage in the lungs caused by foreign viruses. It began mobilizing an army of immune cells to flood into the lungs to destroy viruses and repair damaged tissue.
If a person with Covid-19 infection has a good immune system, the cells will do their duty. They only attack the virus and are stationed in damaged lung areas. The damage is quickly overcome and the person infected with the virus will recover on its own.
However, for people whose immune systems are malfunctioning, their cellular armies simply lose control. They will only flood into the lungs and destroy anything in their path, including viruses, and healthy lung cells.
Therefore, the patient will now suffer double damage from his immune response. The lungs will be more damaged, with more debris, causing blockages and aggravating inflammation.
CT scan of a lung of Covid-19 patient showing lesions in the two lobes.
In the third stage, progressive lung damage can lead to respiratory failure. The scale of the attack is so great that even if the patient survived, these lung lesions would become permanent scars.
Like the previous SARS, Covid-19 could hollow out the lungs, leaving a hole in the hole like a honeycomb. These lesions can be seen on the patient’s x-ray film, the lungs simply white.
The lung will protect itself by forming scars and stiffening. This is the time when the patient can no longer breathe on his own, the doctors must put a breathing machine on them. At the same time, inflammation causes the membranes between the alveoli and blood vessels to become more permeable, causing effusion and limiting the ability of the lungs to absorb oxygen.
“In severe cases, you essentially flood your lungs and can no longer breathe“That’s Frieman. That’s the way Covid-19 kills patients, their own hosts.
* To correctly understand the corona virus that is causing the Covid-19 epidemic, let’s try the following test: